A postdoctoral associate position is available to study HIV-associated neuroinflammation and CNS HIV persistence using advanced humanized mouse models. This position is supported by a newly funded NIH award focused on targeting the NLRP3 inflammasome as a driver of neuroinflammation, blood–brain barrier dysfunction, and latent HIV reservoirs in the central nervous system. The postdoctoral fellow will lead projects using state-of-the-art humanized mouse models of CNS HIV infection to define how innate immune signaling and inflammasome activation in microglia regulate neuroinflammation, BBB integrity, and HIV latency under antiretroviral therapy.
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Education Level
Ph.D. or professional degree